Dear scientists, students and other source code interested

Due to the large amount of requests for help and my limited time, please read the following remarks before asking my assistance:
Please feel free to contact me on scientific questions related to the models and source code, or for bug reports. I will try anwering to those requests and remarks as soon as possible.
However, before contacting me, please do read the articles describing the models carefully, look at the comments in the source code explaining some typos in the articles and hence parameter differences between article and source code and look at the FAQ section first.
Furthermore, please note that I can not answer requests for help with programming problems.
Additional information on my models can be found at the CellML model. repository

C++ source code for the first version of our human ventricular cell model. (ten Tusscher KH, Noble D, Noble PJ, Panfilov AV. A model for human ventricular tissue. Am J Physiol Heart Circ Physiol. 2004;286(4):H1573-89) The model is based on human based experimental data for the individual ionic currents (INa, Ito, IKr, IKs, IK1, ICaL), action potential shape, duration, restitution, and ionic concentration changes. A Java applet of the human ventricular cell model. The applet was developed by Flavio Fenton and Elizabeth Cherry from Hofstra University .

C++ source code for the second version of our human ventricular cell model. (ten Tusscher KH, Panfilov AV. Alternans and spiral breakup in a human ventricular tissue model. Am J Physiol Heart Circ Physiol. 2006; 291: H1088-H1100) Changes relative to the first model: more extensive and realistic description of intracellular calcium dynamics, including subspace calcium dynamics fast and slow voltage inactivation gate in ICaL reproduction of experimentally measured APD restitution curves C++ source code for the reduced version of our human ventricular cell model. (ten Tusscher KH, Panfilov AV. Cell model for efficient simulation of wave propagation in human ventricular tissue under normal and pathological conditions. Phys. Med.Biol., 51: 6141-6156) Changes relative to the full ionic model: no intracellular ion dynamics steady state approximations of fast gating variables adjusted formulation for ICaL